Guy's Gotta Talk About Diabetes, Breast cancer, and Alzheimer's

Sunday, June 30, 2024

DIABETES RESEARCH RIGHT NOW! #19: After Fasting, Why the @!&%$ Are My Blood Sugars STILL Through the ROOF???

From the first moment I discovered I had been diagnosed with DIABETES, I joined a HUGE “club” that has been rapidly expanding since it stopped being a death sentence in the early 20th Century. Currently, there are about HALF A BILLION PEOPLE who have Type 2 Diabetes. For the past 3500 years – dating back to Ancient Egypt – people have suffered from diabetes. Well, I’m one of them now… Not one to shut up for any known reason, I added a section to this blog…

Every month, I’ll be highlighting Diabetes research that is going on RIGHT NOW! Harvested from different websites, journals and podcasts, I’ll translate them into understandable English and share them with you. Today: “One of the most confusing aspects for patients with type 2 diabetes is that we have high fasting glucose levels.” So – WHAT’S THE ANSWER???

“The World Health Organization (WHO) considers type 2 diabetes, one of the pandemics of the 21st century…it is a condition that results in high levels of circulating glucose -- the cellular energy fuel -- due to a deficient insulin response in the body…When blood glucose, also called blood sugar, levels rise after you eat, your pancreas [is supposed to] releases insulin into the blood. Insulin then lowers blood glucose to keep it in the normal range…In T2 patients, the glucose synthesis pathway in the liver (gluconeogenesis) is hyperactivated, a process that can be controlled by drugs such as metformin. ”

But metformin has never CURED T2. It wasn’t meant to. It’s a way to CONTROL T2. This group of researchers noticed something odd. From the beginning of the COVID 19 pandemic, “…factors involved in the control of gluconeogenesis [which is the liver making blood sugars and injecting them into our bloodstream]… sometimes patients hospitalized with COVID-19 showed high glucose levels…[it] seems to be related to the ability of the virus to spark the activity of proteins involved in starting the liver up making glucose and sending it to the bloodstream…”

So, that response of the bodies of people with COVID sometimes made their body react as if they were also Type 2 diabetic. Now, I take FOUR metformin tablets every day. I was SHOCKED to read this: “The mechanisms of action of metformin, the most commonly prescribed drug for the treatment of type 2 diabetes, which reduces how much glucose is in the blood, are still not fully understood.”

In other words, researchers, doctors, and pharmacists DON’T KNOW HOW METFORMIN WORKS!!!!!!! While I intentionally made that more alarm than I could have, the fact is that as researches dig into the method of metformin function, they’re finding out WAY more than they expected, up to and including the effect of metformin in REDUCING AGING!!! “Early evidence highlighted the liver as the major organ involved in the effect of metformin on reducing blood levels of glucose. However, increasing evidence points towards other sites of action that might also have an important role, including the gastrointestinal tract, the gut microbial communities and the tissue-resident immune cells.”

“At the molecular level, it seems that the mechanisms of action vary depending on the dose of metformin used and duration of treatment. Initial studies have shown that metformin targets hepatic mitochondria; however, the identification of a novel target at low concentrations of metformin at the lysosome surface might reveal a new mechanism of action. Based on the effectiveness and safety records in T2DM, attention has been given to the repurposing of metformin as part of adjunct therapy for the treatment of cancer, age-related diseases, inflammatory diseases, and COVID-19.”

Rest assured, I’ll be poking around this paper more as time goes on!

To briefly recap, it seems that HOW metformin works is even deeper than just at the level of CELLS – but deeper still into the “powerhouse” of every cell in your body: the mitochondria…

So, that’s it for now. Obviously I’ll continue to dig deeper into HOW metformin works. (I commented to my wife with a sigh, “Oh, great, now metformin with become as hard to get as Ozempic because the rich will start to pop the pills to stay young and beautiful!”

*sigh* I vote we should just suppress THAT little bit of information!

Links: https://www.sciencedaily.com/releases/2024/06/240607121434.htm; https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4214027/; https://fagron.com/news-media/post/exploring-the-multifaceted-benefits-of-metformin-hydrochloride-beyond-diabetes-management/ ; https://www.nature.com/articles/s41574-023-00833-4

Image: https://asploro.com/wp-content/uploads/2019/12/Diabetes-Research_Open-Access.jpg

Sunday, June 16, 2024

GUY’S GOTTA TALK ABOUT…TYPE 2 DIABETES #22: Getting Older With Type 2

For the first time since I started this blog eleven years ago, it’s going to be about me. I was diagnosed with Type 2 Diabetes two weeks ago. While people are happy to talk about their experiences with diabetes, I WASN’T comfortable with talking about diabetes. My wife is Type 2, as are several friends of ours. The “other Type” of diabetes was what caused the death of my Best Man a year after my wife and I got married. He was diagnosed with diabetes when he was a kid. It was called Juvenile Diabetes then. Today it’s Type 1. Since then, I haven’t WANTED to talk about diabetes at all. But…for my own education and maybe helping someone else, and not one to shut up for any known reason, I’m reopening my blog rather than starting a new one. I MAY take a pause and write about Breast Cancer or Alzheimer’s as medical headlines dictate; but this time I’m going to drag anyone along who wants to join my HIGHLY RELUCTANT journey toward better understanding of my life with Type 2 Diabetes. You’re Welcome to join me!

I was diagnosed with Type 2 diabetes in 2022, so it’s become a “part of who I am”.

I’m gonna confess it right here – I haven’t changed in most ways. I DO exercise more (I bike, on average of three miles a week, between nine and 21 miles a week – depending on how much time I have, the temperature, windiness, rain, and even if I feel like it or not! Even so, my accumulated miles over the past two years went past 1000 a couple weeks ago! AMAZING!

However, I still do Dairy Queen occasionally; I eat peanut-butter/better roll ups for lunch sometimes; eat cookies, candy, and other stuff that’s not good for me…OTOH, I DO watch my diet better than I ever did before my diagnosis. I’m more aware of how foods and exercise affect me; so overall, I’m more cautious about what I eat and how much I exercise than I EVER was in my entire life!

I’m aware of my blood pressure as well. Research has clear connections between diabetes and high blood pressure: “High blood pressure can lead to many complications of diabetes: eye and kidney disease; heart and circulation problems; damages arteries and makes them targets for hardening, called atherosclerosis. That can cause high blood pressure, which if not treated, can lead to trouble including blood vessel damage, heart attack, and kidney failure; coronary artery disease or heart disease; stroke; peripheral vascular disease; hardening of the arteries in the legs and feet; heart failure. Even elevated blood pressure that's at the higher end of normal (120/80 to 129/80) [!!!! My current 20 day BP average is 115/82 – and the bottom one, “diastolic” is more important than the top one “systolic”] impacts your health. Studies show that you have a two to three times greater chance of getting heart disease over 10 years.”

So – how does my upward creeping age affect my diabetes?

“Many of the things you do for your diabetes will also help with high blood pressure:

Control your blood sugar – working on it.
Stop smoking – never did.
Eat healthy – I try…
Exercise most days – yes.
Keep your weight in a healthy range – what the heck is THAT??? Ideal weight 177-188…in the name of perfect transparency: I’ve been 250 +/- for a LONG TIME…
Don't drink a lot of alcohol – don’t do this AT ALL…never have.
Limit how much salt you eat – lately I’ve been experiencing water retention (one of the bits of advice in the article referenced below “Your symptoms may be different”. I’ve never experienced water retention – I suppose that’ll be a great subject for my next “GGTA: Diabetes”…
Visit your doctor regularly – done

With that, I bid you adieu…

Source: https://www.healthline.com/health/type-2-diabetes/changes-after-age-50#changing-symptoms , https://www.webmd.com/diabetes/high-blood-pressure
Image: https://www.hcd.com/wp-content/uploads/2021/01/living-well-with-diabetes.jpg

Sunday, June 2, 2024

DIABETES RESEARCH RIGHT NOW! #18: Researchers Discover New, Safer Treatment Using An OUTDATED Diabetes Drug!

Therefore, in the sight of Americans, TZDs were EVIL! HORRIBLE! USELESS! We want our drugs to do everything – including the laundry and make Big Macs calorie free and make beer non-alcoholic and calorie-free (but not different in ANY other way)

Drugs like TZD and others related to it provide other benefits: “…anti-inflammatory and anti-cancer properties…slow the progression…of coronary hyperplasia ( = “the enlargement of an organ or tissue caused by an increase in the reproduction rate of its cells, often as an initial stage in the development of cancer.”)…beneficial effects on endothelial function, atherogenesis, fibrinolysis, and ovarian steroidogenesis ( = “the processes by which cholesterol is converted to steroid hormones”.)

There are several undesirable side effects to thiazolidinediones, particularly with long-term use. The risks versus benefits require discussion with patients, and alternative first-line agents (metformin, using insulin injections) attempted before using TZDs.

But they are very specific and not common.

Edema and Congestive Heart Failure: (20% of patients; lower doses decrease the risk of edema and weight gain); NOT useful for people who have CHF)

Weight Gain and Fluid Retention: TZDs expand fat tissue mass and can increase weight. Fat gain occurs primarily in tissues just under the skin, sparing the belly area.

Fractures: increased risk and decreased bone density, and are most likely in the forearm, wrist, ankle, foot, tibia, rather than in the hip, pelvis, femur).

Bladder Cancer: One of the TZDs has, in some studies, shown correlations with an increased risk of bladder cancer. However, the most recent analyses do not support an increased risk, and only one of the TZDs increased the risk.

Diabetic Macular Edema: IF an individual takes both the TZDs and regularly use insulin, there may be an increased incidence of diabetic macular edema at 1-year and 10-year follow-up. (Macular edema happens when blood vessels leak into a part of the retina, making it swell, causing blurry vision.)

Increased Ovulation and Teratogenic Effects: This may result in ovulation in some premenopausal women, leading to improved rates of spontaneous pregnancy, but TZDs have some fetal abnormality potential.

So, this research led to a way AROUND using the original TZDs in a totally new way.

How? When fat is inflamed, macrophages release tiny bits of instructions to the cells called microRNAs, small fragments of genetic material that help control what the DNA does for the surrounding cells. These are called exosomes. Released into the bloodstream, they’re picked up by the liver and muscles. This can then lead to changes in obesity and insulin resistance – REDUCING BOTH.

The researchers treated a group of obese mice with a TZD drug. Those mice became more sensitive to insulin, but they also gained weight and retained excess fluid.

When the researchers split the exosomes OUT of the fat tissue, they then injected the macrophages into A DIFFERENT group of fat mice and ALL OF THE positive effects of the TZD worked with none of the bad. The researchers were also able to identify the specific microRNA within the exosomes that was responsible for the beneficial metabolic effects of rosiglitazone. This molecule, called miR-690, could eventually be leveraged into new therapies for type 2 diabetes.

One of the researchers noted, “There's plenty of precedent for using microRNAs themselves as drugs, so that's the possibility we're most excited about exploring for miR-690 going forward.”

It’s NOT today, but because of the discontinued use of one drug, researchers have been able to use increasingly sophisticated procedures to turn it into a NEW drug!

Who knows when? But this is certainly “What’s Next!”

Links: https://www.sciencedaily.com/releases/2024/04/240411130116.htm, https://www.ncbi.nlm.nih.gov/books/NBK551656/

Image: https://asploro.com/wp-content/uploads/2019/12/Diabetes-Research_Open-Access.jpg

Sunday, May 19, 2024

GUY’S GOTTA TALK ABOUT…TYPE 2 DIABETES #22: WHAT Does “Exercise” ACTUALLY Mean?

I chose this image because for most of my life, that’s how I defined “exercising”:

Let’s look at the WORD itself: exorcise (v.) c. 1400, "to invoke spirits," from Old French exorciser (14c.), from Late Latin exorcizare, from Greek exorkizein "banish an evil spirit (or fatness); bind by oath" (see exorcism).

Oops!!! That’s not EXERCIZE – that’s EXORCISE!!! Like the old/new movie!

Hmmm…truth be told, I have about as much interest in exercising as I taking part in an exorcism…Not even sure how much difference there is between them, to be honest.

At any rate, exercise carries so much weight (pun originally NOT intended, but let’s roll with it), in my mind that even the mention of it or seeing commercials with people exercising in them brings me out in a cold sweat. NOT a hot sweat, so worrying about exercising isn’t gonna do me any good.

Lemme get back on track, I go to an etymological website (no, NOT a bug website!) to explore the ORIGINS of words. In this case, the word exercise has these roots: “…condition of being in active operation; practice for the sake of training," from Old French exercice.”

I’m going to change one word in there – not really change it, but at two letters. Where it says, “…condition of being in active operation; practice….blah, blah, blah”.

I’m going to amend that to a “…condition of being in active COOPERATION…” because you have to cooperate with others if you are actually going to exercise…I suppose you can go it alone, but bringing a cheer squad with you is helpful!

I’m NOT talking about the adolescent acrobatics depicted in the picture above. What I’m THINKING about is the image at the top of the column.

Just start walking…biking…gardening…cleaning up a nearby park…counting hummingbirds for your local or state County or State Park Reserves…band migratory birds or even just COUNT them for population studies. Walk in the winter, too! You don’t have to walk miles – in the winter I started by walking from our house to the graveyard at the top of the hill (TALK ABOUT MOTIVATION!!!) Moving is a good thing. ANY AMOUNT OF MOVING CAN BE GOOD. I’ve said it before and I’ll keep on saying it. So if you don’t want to hear this “moving stuff”, just don’t read my blogs if they have the word “move” in it…

A reasonable read about the benefits of exercise from the Mayo Clinic’s website: https://www.mayoclinic.org/healthy-lifestyle/fitness/in-depth/exercise/art-20048389
Image: https://cathe.com/wp-content/uploads/2017/07/shutterstock_410678884.jpg Image: https://www.hcd.com/wp-content/uploads/2021/01/living-well-with-diabetes.jpg

Sunday, May 5, 2024

DIABETES RESEARCH RIGHT NOW! #17: Why Do Some Obese People Get Type 2 and Some DON’T? BROKEN CLEANING CELLS!

From the first moment I discovered I had been diagnosed with DIABETES, I joined a HUGE “club” that has been rapidly expanding since it stopped being a death sentence in the early 20th Century. Currently, there are about HALF A BILLION PEOPLE who have Type 2 Diabetes. For the past 3500 years – dating back to Ancient Egypt – people have suffered from diabetes. Well, I’m one of them now… Not one to shut up for any known reason, I added a section to this blog…

Every month, I’ll be highlighting Diabetes research that is going on RIGHT NOW! Harvested from different websites, journals and podcasts, I’ll translate them into understandable English and share them with you. Today: STARTING with a 2014 article, “Diabetes – Will it Ever be cured?”; I check up on the various therapies mentioned…

If I were to ask you what YOU think causes Type 2 diabetes in people…OK, how about I just ask myself?

“Guy, what do you think caused your Type 2 diabetes?”

I’d answer, “I’m fat.”

And I’d be right – but not ENTIRELY! How is it that I’m Type 2 and someone else I know who is JUST as fat NOT Type 2 diabetic????

“It’s just not FAIR!” I wail, weeping, pounding my chest and “Woe-ing-is-me-ing!” to beat the band.

Well, Mr. Me, I don’t know about fairness in biological problems, challenges, and illnesses – but I DO know that science is hard at work at figuring our a way to stop Type 2 from happening; fixing it; or curing it altogether! It’s an expensive disease that is spreading over the entire planet!

“Scientists and physicians have been documenting the condition now known as diabetes for thousands of years.” “in 1675 the word ‘mellitus’, meaning honey, was added to the name ‘diabetes’, meaning siphon. It wasn't until the 1800s that scientists developed chemical tests to detect the presence of sugar in the urine.”

In 1888, a French physician believed that diabetics were either thin or fat. The fat ones were ALWAYS rich and fat and had what was called “adult onset” diabetes. In contrast, thin diabetics always thin, young, desperately ill and, within a year, dead of what was called “adolescent onset” diabetes. Adult Onset was a disease of wealth and laziness; secondary to poor digestion and bad nerves. If you had it, you would die early, albeit you had longer than twelve months to live! The introduction of insulin in 1921 commuted the death sentence to a life sentence but emphasized the difference between insulin-dependent diabetes or adolescent-onset and non-insulin-dependent diabetes. Today, it’s Type 1 and Type 2 diabetes. Rates of type 2 diabetes have increased since 1960 unsurprisingly right along with obesity.

In 1985, there were 30 million Type 2 diabetics in the US by 2015 there were some 392 million people who were Type 2; by 2030, they expect some 600,000,000 people will be diagnosed as Type 2. But how is it that some people who gain weight suffer from the disease and others do not. The reasons for these differences are not clear, but they are related to what you body USES the fat it gets for rather than HOW MUCH body fat your body has.

The research below noticed that “Healthy fat tissue protects against new fat getting in deposited in the WRONG places. Human beings ALL need fat! The main purpose is that fat is laid down DIRECTLY underneath the skin. THAT fat protects the body from injury.

But it serves a second job: when you need energy, fat breaks down into sugar and goes to the cells so that can “burn it” to keep running. In PARTICULAR, muscle cells need sugar to keep you moving. You’re sleeping at night, so you don’t need as much instant energy, so the body stuffs your LIVER full of fat just waiting to be broken down and used as energy.

Most of you have heard the word “collagen” and you probably know that collagen is a protein in the body. Different kinds of collagen show up in many body parts like hair, skin, fingernails and toenails, bones, ligaments, tendons, cartilage, blood vessels, and intestines. IOW: It’s IMPORTANT!

Your body MAKES fat to story energy, right? The body breaks down collagen to build up fat reserves.

Scientists have found that the rise in collagen breakdown during adipose tissue expansion is done by large microscopic cells called “macrophage” (it literally means “big eater”! However, if you get OVERLY FAT, the macrophages BREAK, losing their ability to turn fat cells into energy ready for the body to use. The broken pieces of collagen are not just waste products, but they stimulate the making of MORE macrophages. That causes inflammation effects in the cells.

The most amazing discovery was still to come. Those macrophages and collagen fragments.: IT APPEARS LIKE if we can TARGET them, we JUST MIGHT BE ABLE TO PREVENT Type-2 diabetes and other conditions of impaired tissue remodeling!”

In science language: “In conclusion, this study highlights the importance of collagen-degrading macrophages and efficient removal of collagen fragments in adaptive, weight gain–induced adipose tissue remodeling. Our data suggest that impaired macrophage-mediated intracellular collagen degradation in obese SAT cannot be fully compensated for by extracellular collagen degradation. We conclude that collagen fragments, rather than being inert metabolites and solely markers of tissue remodeling, actively participate in shaping the SAT microenvironment. Further research in this area may identify novel targets in the prevention of type-2 diabetes in subjects with obesity and in other areas of impaired tissue remodeling.”

In plain English? Not ONLY can macrophages themselves help prevent Type-2; but the FRAGMENTs of the collagen from that the macrophages made when they attacked collagen, just might help as well.

Links: https://www.sciencedaily.com/releases/2024/02/240201212856.htm; https://www.pnas.org/doi/10.1073/pnas.2313185121 ; https://www.ncbi.nlm.nih.gov/pmc/articles/PMC6946720/ Image: https://asploro.com/wp-content/uploads/2019/12/Diabetes-Research_Open-Access.jpg

Sunday, April 21, 2024

GUY’S GOTTA TALK ABOUT…TYPE 2 DIABETES #21: How To Happily Eat Summer – WITHOUT Your Glucose Hitting 600!

For the first time since I started this blog eleven years ago, it’s going to be about me. I was diagnosed with Type 2 Diabetes two weeks ago. While people are happy to talk about their experiences with diabetes, I WASN’T comfortable with talking about diabetes. My wife is Type 2, as are several friends of ours. The “other Type” of diabetes was what caused the death of my Best Man a year after my wife and I got married. He was diagnosed with diabetes when he was a kid. It was called Juvenile Diabetes then. Today it’s Type 1. Since then, I haven’t WANTED to talk about diabetes at all. But…for my own education and maybe helping someone else, and not one to shut up for any known reason, I’m reopening my blog rather than starting a new one. I MAY take a pause and write about Breast Cancer or Alzheimer’s as medical headlines dictate; but this time I’m going to drag anyone along who wants to join my HIGHLY RELUCTANT journey toward better understanding of my life with Type 2 Diabetes. You’re Welcome to join me!

If you want to read more, just click on the three articles below for great information about ways to have fun WHILE keeping your Type 2 in good standing.

First of all, let me tell you, I am NOT a Poster Old Man for smart Type 2 Diabetes living. Staying away from foods I love is often difficult, and I sometimes just give in and go to DQ anyway. But I haven’t given up entirely. We still try to eat right and avoid certain foods. Friends of our help to keep us on the “straight-and-narrow” by their own personal choices (BUT NOT BY PREACHING!) That’s what I try and do here – I try to share what things that work for me.

One example is exercising. I hate exercise. There, I said it.

HOWEVER, I love riding my bike. I’ve liked biking since my parents bought me a three-speed for a Confirmation gift. That started a habit that’s lasted over half a century. Of course, shortly after my parents got me the three-speed, I got a job and bought a REAL bike: my first “curly-handle ten speed”. I’ve been riding ever since. I even use my bike in the winter. A friend of mine lent me his indoor bike stand. For the past three winters I’ve spent about half an hour every other day pedaling away (in boredom), while waiting for Spring to come; or riding on the trails as long as could into Fall.

During the summer, I rotate five different (and hopefully finally add a sixth – two times!) trails. Their LENGTH isn’t as different as that two of them force me to climb a ramp over a busy highway.

At any rate, the three articles I reference below deal mostly with EATING over the summer months! Boiling down TWO of them (the other is Diabetic-Friendly recipes), give me this lists of things I should either CONTINUE doing or try starting.

If you use insulin, BEWARE: heat makes the insulin absorb into your skin faster; it can also be damaged by high temperatures; also, heat alters the accuracy of your glucose monitors -- and even the test strips. Just be aware of where and how you carry your "gear".

A) Try NOT to sit around at summer parties -- move around, chat with people, even volunteer to bring foods in and out, or bring cans of pop out.

B) Be aware of how fluid Summer-eating times can be! Lunch can easily slide to supper on a hot day, and the evening meal can show up after 10 pm! We don't have the luxury of a stable metabolism that can handle eating a box of Dunkin Donuts after midnight!

C) Don't load up on carbs and sugar after the sun goes down! You might be alarmed at your glucose readings when you check them first thing in the morning like I do!

D) Don't power load on carbs! (This is me reminding myself, I'm sure you have better control than I do!!!)

E) Sit down and eat a plateful rather than grazing the tables and bags of chips and dips and coolers and...well, you know what I mean! I'm going to try next week at my nephew's wedding: HALF the plate is veggies and meats; the other half can be carbs: buns, cakes, cookies, candies. Keep that balance! (I might try (I) as well...)

F) If possible, pick the grilled chicken breast rather than the grilled burger -- oh, and use half a bun instead of the whole thing -- in either case!

G) If you're eating with close family: ASK FOR OPTIONS! They love you (presumably) but as non-Type Twos, they don't THINK OF HAVING TO CHOOSE healthy options! They WANT you to be around to change diapers and go to concerts and basketball games!

H) DON'T SKIP MEALS BEFORE THE BIG BBQ!!!! I try not to do that anymore! Or if I do, I work extra hard to balance the "big meal" like the plate up in E.

I) Grab the nearest loved one you haven't seen for a while and take a survey of the SPREAD with them noting what's there, what's not there, and what you look forward to eating! Being AWARE of our eating is more important than just about ANYTHING we can do to remain healthy while being happy, too!

Lastly, bring a HEALTHY DISH you can share – scan the recipe website below for suggestions. Even the most sugar-addled, carbohydrate magnet young adult might be open to trying something new and tasty (make SURE you test the recipes before you bring them!) The majority won’t even notice if your deviled eggs have egg whites substituting for HALF the usual six yolks; mixed in low-fat mayo; and checking the mustard content – just skip adding the half cup of sawdust most people assume low-carb, low fat foods will taste like!

Lastly, have fun! Take the grandkids to the park at the end of the street; go for a walk about the block after the meal (better yet, BEFORE the meal!)

These are mostly easy things! I’m certainly going to deploy them in defending my body against the ravages of Type 2!

Source: https://www.diabetes.org.uk/guide-to-diabetes/enjoy-food/cooking-for-people-with-diabetes/seasonal-cooking/summer-eating-and-diabetes,
Summer Type Two Dining: https://www.eatingwell.com/gallery/7968046/easy-summer-diabetes-friendly-dinners/#:~:text=Dinners%20with%20bright%20ingredients%20from,a%20diabetes%2Dfriendly%20eating%20pattern.
Image: https://www.hcd.com/wp-content/uploads/2021/01/living-well-with-diabetes.jpg

Sunday, April 7, 2024

DIABETES RESEARCH RIGHT NOW! #16: The ARTIFICIAL PANCREAS – Today for Type 1; Tomorrow for Type 2!

https://www.usmeddirect.com/cdn/shop/products/FreeStyleLibre2_002_002_grande.png?v=1597075020

Linked to a blood monitor sensor, that is linked in with an insulin delivery system – hence it’s a closed loop. The person with the “artificial pancreas” doesn’t have to take their blood sugars, then inject insulin themselves. It’s done automatically.

Amazing. Even more amazing that “the first experimental artificial pancreas was developed in 1964.” From that first step to a viable artificial pancreas that was invented for people with Type 1 diabetes…and now applicable to people with Type 2 – all in sixty years!

YOU may be the next recipient of an ARTIFICIAL PANCREAS!

Links: https://www.healthline.com/health-news/type-2-diabetes-an-artificial-pancreas-may-help-improve-blood-sugar-levels , https://myacare.com/blog/artificial-pancreas-technology-for-diabetes-management-types-and-benefits, https://finance.yahoo.com/news/artificial-pancreas-device-systems-market-230200881.html?guccounter=1&guce_referrer=aHR0cHM6Ly93d3cuZ29vZ2xlLmNvbS8&guce_referrer_sig=AQAAADBAy9cdsnafVvSQuS0pjYfdIf-U4seLpKhN_HaLqwEmE_0I19d11FV250q3MVWSWqKoB-9VWDfxSQtxXYhRmVHjKDjbB_k8BKxOnSV24tVKvwmXQWy2Tkke_bDQTjrbTWj4mlX65TvZrYTt4LH4BhqfFREeFtEX0fNpMNDV2Bfo , https://www.fda.gov/medical-devices/recently-approved-devices/minimed-780g-system-p160017s091 , https://www.niddk.nih.gov/news/archive/2017/story-discovery-artificial-pancreas-managing-type1-diabetes
Image: https://asploro.com/wp-content/uploads/2019/12/Diabetes-Research_Open-Access.jpg